Diffuse alveolar haemorrhage

CT

First and second year — the floor first, then every step

Bilateral ground glass and consolidation with centrilobular nodules and, days later, crazy paving — with haemoptysis, a falling haemoglobin and a vasculitis or anticoagulation context; BAL with sequentially bloodier returns confirms it.

Orient first

  • Causes: ANCA vasculitis, anti-GBM disease, SLE, anticoagulation, drugs, bone marrow transplant.
  • Haemoptysis is absent in a third; the imaging and a haemoglobin drop may be the first clue.
  • Clearing within days separates it from infection and oedema.

Acquire the study

  • Non-contrast CT chest with thin sections.

The manoeuvre

  • Axial lung window: bilateral ground glass and consolidation — distribution usually central and perihilar, sparing the periphery.
  • Centrilobular ground-glass nodules.
  • Crazy paving (septal thickening over ground glass) after 2–3 days.
  • Effusions and nodes minimal; compare with prior study for rapid change.
  • Kidneys or sinuses if imaged (pulmonary-renal syndromes).

What confirms it

  • Progressively haemorrhagic BAL returns with haemosiderin-laden macrophages and a compatible CT.

What licenses you to exclude it

  • CT cannot exclude small-volume haemorrhage; BAL can.

The classic misread

  • Calling it pneumonia and missing the vasculitis.
  • Calling crazy paving alveolar proteinosis without the clinical context.

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